By Robert Klein, George Enders
This one of a kind attempt prep advisor allows you to try out your wisdom of crucial anatomy, histology and telephone biology thoughts for the USMLE Step 1; perform with 500 USMLE Step 1-style questions with referenced solutions; evaluate factors for correct and mistaken answers;and construct self assurance, abilities, and data.
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Extra info for Anatomy, Histology, and Cell Biology PreTest Self-Assessment and Review
In Neural Mechanisms of Conditioning (eds D. L. Alkon and C. D. Woody), pp. 397-420. Plenum Press, New York and London. Levitan, I. B. and Benson, J. A. (1981) Neuronal oscillators in Aplysia: modulation by serotonin and cyclic AMP. Trends NeuroSci. 4, 38-41. Prince, D. A. (1978) Neurophysiology of epilepsy. Annu. Rev. Neurosci. 1, 395-415. Rinzel, J. and Lee, Y. S. (1987) Dissection of a model for neuronal parabolic bursting. /. Math. Biol. 25, 653-75. Schwartzkroin, P. A. and Stafstrom, C. E.
The positive overshoot of the action potentials increases to midburst and then remains fairly constant. The undershoot (negative afterpotential) decreases almost throughout the burst. At the end of the burst there is a slow depolarizing after-potential (DAP) followed by the post-burst hyperpolarization. The hyperpolarization decays during the interburst inter val at an increasing rate leading to a depolarizing inflection that triggers the first action potential of the next burst. The following description of the ionic mechanism of bursting probably applies to many but not all molluscan bursters.
A second approach questions the legitimacy of the foundation of the Ca 2+ hypothesis, namely that Ca 2+ is the only limiting factor in the release process. It suggests that while Ca 2+ is certainly required for release, it is nevertheless unable to support evoked release by itself. This approach assumes that another factor, together with Ca 2+ , accumulates during the natural stimulus, and that both this factor and Ca 2+ are required to start the chain of events leading to release. In the case of the neuromuscular junction, the natural stimulus is membrane depolarization, and therefore this second factor, called S, must be produced by membrane depolarization and must rapidly disap pear with membrane repolarization.